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nikon biostation ct  (Nikon)


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    Structured Review

    Nikon nikon biostation ct
    Nikon Biostation Ct, supplied by Nikon, used in various techniques. Bioz Stars score: 95/100, based on 272 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/biostation+ct/BioStation+CT/pmc12854672-133-21-21
    Average 95 stars, based on 272 article reviews
    nikon biostation ct - by Bioz Stars, 2026-09
    95/100 stars

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    Related Articles

    other:

    Article Title: Swift induction of human spinal lower motor neurons and robust ALS cell screening via single-cell imaging
    Article Snippet: Images were acquired using BioStation CT (Nikon) from day 3 to day 13.


    Article Title: Morphology and biological characteristics of the electrochemically modified titanium surface.
    Article Snippet: An anodic polarization of the commercially pure titanium samples in 0.1 M phosphate buffer solution (pH 7.4) in the range of potentials 0.00 to 1.00 V provides an electrochemical surface modification due to controlled passivation.. The titanium oxide film thickness calculated using electrochemical impedance spectroscopy falls in two ranges: 8–11 nm for experiments conducted at open circuit potentials, and 12–26 nm for experiments under conditions of anodic polarization.. The maximum value of the root mean square roughness (Ra = 80 nm) was obtained using a polarization potential of 0.75 V. At the same potential, minimal interaction of rat’s fibroblasts with the titanium samples surface was observed.

    Imaging:

    Article Title: Mitochondrial Intercellular Transfer via Platelets After Physical Training Exerts Neuro‐Glial Protection Against Cerebral Ischemia
    Article Snippet: .. Mitochondrial uptake into various cells was evaluated using a BioStation CT (Nikon, Tokyo, Japan) incubator equipped with a camera for video imaging. ..

    Article Title: Swift induction of human spinal lower motor neurons and robust ALS cell screening via single-cell imaging.
    Article Snippet: .. Automated time-lapse live imaging Images were acquired using BioStation CT (Nikon) from day 3 to day 13. ..

    Article Title: Swift induction of human spinal lower motor neurons and robust ALS cell screening via single-cell imaging
    Article Snippet: .. Automated time-lapse live imaging Images were acquired using BioStation CT (Nikon) from day 3 to day 13. ..

    Microscopy:

    Article Title: Determination and validation of design space for mesenchymal stem cell cultivation processes using prediction intervals
    Article Snippet: .. Phase contrast microscopy images were automatically acquired every 6 h via BioStation CT (Nikon Corporation, Tokyo, Japan) at 4× magnification (8 × 8 tiling per well, 15.3 × 15.3 mm, 1000 pixels/image). ..

    Cell Culture:

    Article Title: Vascularized Bioengineered Kidney Using Decellularized Scaffold Recellularized with human Placenta-Derived Angiogenic stem Cells and Kidney Organoids
    Article Snippet: .. hPASCs at passage 3 were cultured in 3D Matrigel and imaged using a biostation CT (Nikon) at 37°C with 5% CO2. ..



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    Generation and functional characterization of highly invasive CT-26 (HI CT-26) cells. ( A ) Schematic workflow for generating HI CT-26 cells. CT-26 cells stably expressing EGFP were injected into mice via the tail vein. After 7 days, liver tumor tissues were isolated, sorted by flow cytometry, and reinjected into recipient mice. This cycle was repeated three times to obtain HI CT-26 cells. ( B , D ) Transwell assay demonstrating enhanced migratory capacity of HI CT-26 cells compared to controls, imaged via bright-field/fluorescence microscopy ( B ), with corresponding quantitative analysis ( D ). ( C , E ) Invasion assay showing significantly enhanced invasive potential of HI CT-26 cells compared to controls, with representative bright-field images ( C ) and corresponding quantitative analysis ( E ). Quantitative analysis of the Transwell migration and invasion assays, respectively. **Data are presented as mean ± SD; n = 3 independent experiments. Statistical significance was determined by unpaired two-tailed Student’s t-test (* p < 0.05, ** p < 0.01). ( F <t>)</t> <t>Live-cell</t> tracking over 24 h revealed prolonged movement trajectories and higher motility in HI CT-26 cells. ( G ) Transendothelial migration assay: HI CT-26 cells crossed the HUVEC endothelial barrier within ~ 6 h, whereas < 60% of control cells completed transmigration within 12 h (see Movies 1–2, which vividly illustrate the accelerated migration speed and distinct morphological dynamics of HI CT-26 cells compared to controls. (H) Western blot analysis of EMT markers: HI CT-26 cells exhibited reduced E-cadherin and elevated N-cadherin, Vimentin, and Snail levels
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    Generation and functional characterization of highly invasive CT-26 (HI CT-26) cells. ( A ) Schematic workflow for generating HI CT-26 cells. CT-26 cells stably expressing EGFP were injected into mice via the tail vein. After 7 days, liver tumor tissues were isolated, sorted by flow cytometry, and reinjected into recipient mice. This cycle was repeated three times to obtain HI CT-26 cells. ( B , D ) Transwell assay demonstrating enhanced migratory capacity of HI CT-26 cells compared to controls, imaged via bright-field/fluorescence microscopy ( B ), with corresponding quantitative analysis ( D ). ( C , E ) Invasion assay showing significantly enhanced invasive potential of HI CT-26 cells compared to controls, with representative bright-field images ( C ) and corresponding quantitative analysis ( E ). Quantitative analysis of the Transwell migration and invasion assays, respectively. **Data are presented as mean ± SD; n = 3 independent experiments. Statistical significance was determined by unpaired two-tailed Student’s t-test (* p < 0.05, ** p < 0.01). ( F <t>)</t> <t>Live-cell</t> tracking over 24 h revealed prolonged movement trajectories and higher motility in HI CT-26 cells. ( G ) Transendothelial migration assay: HI CT-26 cells crossed the HUVEC endothelial barrier within ~ 6 h, whereas < 60% of control cells completed transmigration within 12 h (see Movies 1–2, which vividly illustrate the accelerated migration speed and distinct morphological dynamics of HI CT-26 cells compared to controls. (H) Western blot analysis of EMT markers: HI CT-26 cells exhibited reduced E-cadherin and elevated N-cadherin, Vimentin, and Snail levels
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    Generation and functional characterization of highly invasive CT-26 (HI CT-26) cells. ( A ) Schematic workflow for generating HI CT-26 cells. CT-26 cells stably expressing EGFP were injected into mice via the tail vein. After 7 days, liver tumor tissues were isolated, sorted by flow cytometry, and reinjected into recipient mice. This cycle was repeated three times to obtain HI CT-26 cells. ( B , D ) Transwell assay demonstrating enhanced migratory capacity of HI CT-26 cells compared to controls, imaged via bright-field/fluorescence microscopy ( B ), with corresponding quantitative analysis ( D ). ( C , E ) Invasion assay showing significantly enhanced invasive potential of HI CT-26 cells compared to controls, with representative bright-field images ( C ) and corresponding quantitative analysis ( E ). Quantitative analysis of the Transwell migration and invasion assays, respectively. **Data are presented as mean ± SD; n = 3 independent experiments. Statistical significance was determined by unpaired two-tailed Student’s t-test (* p < 0.05, ** p < 0.01). ( F <t>)</t> <t>Live-cell</t> tracking over 24 h revealed prolonged movement trajectories and higher motility in HI CT-26 cells. ( G ) Transendothelial migration assay: HI CT-26 cells crossed the HUVEC endothelial barrier within ~ 6 h, whereas < 60% of control cells completed transmigration within 12 h (see Movies 1–2, which vividly illustrate the accelerated migration speed and distinct morphological dynamics of HI CT-26 cells compared to controls. (H) Western blot analysis of EMT markers: HI CT-26 cells exhibited reduced E-cadherin and elevated N-cadherin, Vimentin, and Snail levels
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    Generation and functional characterization of highly invasive CT-26 (HI CT-26) cells. ( A ) Schematic workflow for generating HI CT-26 cells. CT-26 cells stably expressing EGFP were injected into mice via the tail vein. After 7 days, liver tumor tissues were isolated, sorted by flow cytometry, and reinjected into recipient mice. This cycle was repeated three times to obtain HI CT-26 cells. ( B , D ) Transwell assay demonstrating enhanced migratory capacity of HI CT-26 cells compared to controls, imaged via bright-field/fluorescence microscopy ( B ), with corresponding quantitative analysis ( D ). ( C , E ) Invasion assay showing significantly enhanced invasive potential of HI CT-26 cells compared to controls, with representative bright-field images ( C ) and corresponding quantitative analysis ( E ). Quantitative analysis of the Transwell migration and invasion assays, respectively. **Data are presented as mean ± SD; n = 3 independent experiments. Statistical significance was determined by unpaired two-tailed Student’s t-test (* p < 0.05, ** p < 0.01). ( F <t>)</t> <t>Live-cell</t> tracking over 24 h revealed prolonged movement trajectories and higher motility in HI CT-26 cells. ( G ) Transendothelial migration assay: HI CT-26 cells crossed the HUVEC endothelial barrier within ~ 6 h, whereas < 60% of control cells completed transmigration within 12 h (see Movies 1–2, which vividly illustrate the accelerated migration speed and distinct morphological dynamics of HI CT-26 cells compared to controls. (H) Western blot analysis of EMT markers: HI CT-26 cells exhibited reduced E-cadherin and elevated N-cadherin, Vimentin, and Snail levels
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    Image Search Results


    Generation and functional characterization of highly invasive CT-26 (HI CT-26) cells. ( A ) Schematic workflow for generating HI CT-26 cells. CT-26 cells stably expressing EGFP were injected into mice via the tail vein. After 7 days, liver tumor tissues were isolated, sorted by flow cytometry, and reinjected into recipient mice. This cycle was repeated three times to obtain HI CT-26 cells. ( B , D ) Transwell assay demonstrating enhanced migratory capacity of HI CT-26 cells compared to controls, imaged via bright-field/fluorescence microscopy ( B ), with corresponding quantitative analysis ( D ). ( C , E ) Invasion assay showing significantly enhanced invasive potential of HI CT-26 cells compared to controls, with representative bright-field images ( C ) and corresponding quantitative analysis ( E ). Quantitative analysis of the Transwell migration and invasion assays, respectively. **Data are presented as mean ± SD; n = 3 independent experiments. Statistical significance was determined by unpaired two-tailed Student’s t-test (* p < 0.05, ** p < 0.01). ( F ) Live-cell tracking over 24 h revealed prolonged movement trajectories and higher motility in HI CT-26 cells. ( G ) Transendothelial migration assay: HI CT-26 cells crossed the HUVEC endothelial barrier within ~ 6 h, whereas < 60% of control cells completed transmigration within 12 h (see Movies 1–2, which vividly illustrate the accelerated migration speed and distinct morphological dynamics of HI CT-26 cells compared to controls. (H) Western blot analysis of EMT markers: HI CT-26 cells exhibited reduced E-cadherin and elevated N-cadherin, Vimentin, and Snail levels

    Journal: Cancer Cell International

    Article Title: The notch-miR-188-5p-TIMP2/3 axis orchestrates exosome-driven pre-metastatic niche formation in colorectal cancer

    doi: 10.1186/s12935-025-04164-4

    Figure Lengend Snippet: Generation and functional characterization of highly invasive CT-26 (HI CT-26) cells. ( A ) Schematic workflow for generating HI CT-26 cells. CT-26 cells stably expressing EGFP were injected into mice via the tail vein. After 7 days, liver tumor tissues were isolated, sorted by flow cytometry, and reinjected into recipient mice. This cycle was repeated three times to obtain HI CT-26 cells. ( B , D ) Transwell assay demonstrating enhanced migratory capacity of HI CT-26 cells compared to controls, imaged via bright-field/fluorescence microscopy ( B ), with corresponding quantitative analysis ( D ). ( C , E ) Invasion assay showing significantly enhanced invasive potential of HI CT-26 cells compared to controls, with representative bright-field images ( C ) and corresponding quantitative analysis ( E ). Quantitative analysis of the Transwell migration and invasion assays, respectively. **Data are presented as mean ± SD; n = 3 independent experiments. Statistical significance was determined by unpaired two-tailed Student’s t-test (* p < 0.05, ** p < 0.01). ( F ) Live-cell tracking over 24 h revealed prolonged movement trajectories and higher motility in HI CT-26 cells. ( G ) Transendothelial migration assay: HI CT-26 cells crossed the HUVEC endothelial barrier within ~ 6 h, whereas < 60% of control cells completed transmigration within 12 h (see Movies 1–2, which vividly illustrate the accelerated migration speed and distinct morphological dynamics of HI CT-26 cells compared to controls. (H) Western blot analysis of EMT markers: HI CT-26 cells exhibited reduced E-cadherin and elevated N-cadherin, Vimentin, and Snail levels

    Article Snippet: The migration process was dynamically monitored using a live-cell imaging workstation (Nikon BioStation CT) with phase-contrast microscopy and multi-channel fluorescence imaging.

    Techniques: Functional Assay, Stable Transfection, Expressing, Injection, Isolation, Flow Cytometry, Transwell Assay, Fluorescence, Microscopy, Invasion Assay, Migration, Two Tailed Test, Cell Tracking Assay, Control, Transmigration Assay, Western Blot